Does plasma ANP participate in natriuresis induced by a-MSH?

-Melanocyte-stimulating hormone (-MSH; 0.6 and 3 nmol) microinjected into the anteroventral region of the third ventricle (AV3V) induced a significant increase in diuresis without modifying natriuresis or kaliuresis. Intraperitoneal (ip) injection of -MSH (3 and 9.6 nmol) induced a significant in...

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Bibliographic Details
Authors: DINIZ, Domingos Luiz Wanderley Picanço, OLIVEIRA, Gustavo Ribeiro de, FAVARETTO, Ana Lucia Vianna, GUTKOWSKA, J., MCCANN, S.M., RODRIGUES, José Antunes
Format: article
Status:Published version
Publication Date:1997
Country:Brasil
Institution:Universidade Federal do Pará (UFPA)
Repository:Repositório Institucional da UFPA
Language:English
OAI Identifier:oai:repositorio.ufpa.br:2011/3290
Online Access:http://repositorio.ufpa.br/jspui/handle/2011/3290
Access Level:Open access
Keyword:Hormônio estimulador de melanócito
Fator natriurético atrial
Natriurese
Caliureses
Diurelis
Description
Summary:-Melanocyte-stimulating hormone (-MSH; 0.6 and 3 nmol) microinjected into the anteroventral region of the third ventricle (AV3V) induced a significant increase in diuresis without modifying natriuresis or kaliuresis. Intraperitoneal (ip) injection of -MSH (3 and 9.6 nmol) induced a significant increase in urinary sodium, potassium and water excretion. Intraperitoneal (3 and 4.8 nmol) or iv (3 and 9.6 nmol) administration of -MSH did not induce any significant changes in plasma atrial natriuretic peptide (ANP), suggesting that the natriuresis, kaliuresis and diuresis induced by the systemic action of -MSH can be dissociated from the increase in plasma ANP. These preliminary results suggest that -MSH may be involved in a -MSHindependent mechanism of regulation of hydromineral metabolism.